PhD candidate / guest
Yi Ding’s research focuses on understanding how transcriptomics and metabolomics are affected by an obese gene called leptin and mycobacterial infection which results in tuberculosis. For this, zebrafish, as an animal model, and multidisciplinary approaches from biology, chemistry and bioinformatics are used.
Ding, Y., Raterink, R., Marín-Juez, R. et al. Tuberculosis causes highly conserved metabolic changes in human patients, mycobacteria-infected mice and zebrafish larvae. Sci Rep 10, 11635 (2020). https://doi.org/10.1038/s41598-020-68443-y